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Andrew M. Ball PT PhD -> Re: peroneal spasticity (August 27, 2006 3:58:00 PM)
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Emad,
Good point, but I'm of the NDT thinking that, in general, neurologic tone is posturally dependent, and that the UMN lesion's direct effect is that of HYPOTONIA of core musculature, with everything else (e.g. UE and/or LE hypertonia) being an indirect effect.
Under that framework, there is a sequential pattern of increasing tone as we observe distally -- and peroneal "spasticity" usually isn't in the pattern (that's not to say it can't be . . . just unusual). Also, under that framework, "specific muscle spasticity" is now used to describe local muscular abnormalities not associated with an UMN lesion, such as congenital torticollis.
Cross-reference with the fact that the surgery was local to the foot, peroneal area in particular, and we'd want to think horses before zebras. An UMN lesion that was anesthesia related would be MUCH more likely to impact much more than just peroneal musculature (physical findings alone would be much more likely to produce gastroc hypertonicity, clonus, and babinski . . . none of which was described).
I think that, as an outpatient orthopedic PT, Sabby actually used the term correctly to describe specific peroneal muscle spasticity, absent of UML lesion. At least that's how I read it.
Drew
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